Skip to main content
メニュー
Revvity logo
Contact us
JP
Revvity Sites Globally

Select your location.

*e-commerce not available for this region.

australia.webp Australia
austria.webp Austria
belgium.webp Belgium
brazil.webp Brazil *
canada.webp Canada
china.webp China *
denmark.webp Denmark
finland.webp Finland
france.webp France
germany.webp Germany
hong-kong.webp Hong Kong (China) *
india.webp India *
ireland.webp Ireland
italy.webp Italy
japan.webp Japan *
luxembourg.webp Luxembourg
mexico.webp Mexico *
netherlands.webp Netherlands
norway.webp Norway
philippines.webp Philippines *
republic of korea.webp Republic of Korea *
singapore.webp Singapore *
spain.webp Spain
sweden.webp Sweden
switzerland.webp Switzerland
thailand.webp Thailand *
uk.webp United Kingdom
usa.webp United States
Breadcrumb
...
  • ホーム
  • Services
  • Revvity Omics Services
  • Revvity Omics Clinical Services
  • Sponsored Testing Programs
    • Cytogenomics
    • Global Laboratory Network
    • Metabolic Testing
    • Newborn Screening Services
    • Prenatal Screening Services
    • Proactive Testing
    • Rare Disease Testing
    • Specialized and Customized Assays
    • Sponsored Testing Programs
  • The Lantern Project
    • Autoimmune Type 1 Diabetes Early Detection Program
    • Decode Duchenne
    • Roadmap2Rare
    • The Lantern Project
    • Ultragenyx MPS Testing Program
  • Interstitial Lung Disease Panel
    • Fabry Disease
    • Focused Neuromuscular Disease Panel
    • Gaucher Disease & Niemann - Pick Type A & B (ASMD)
    • Interstitial Lung Disease Panel
    • Mucopolysaccharidosis I (MPS I) & Other MPS Disorders
    • Pain and Cerebrovascular Panel
    • Pompe Disease

Interstitial Lung Disease Panel

Interstitial lung disease (ILD) refers to the diverse group of chronic lung disorders characterized by inflammation and scarring within the lung tissue.1 These lung disorders can include familial pulmonary fibrosis, idiopathic pulmonary fibrosis, and progressive pulmonary fibrosis.2 Fibrosis leads to stiff lung tissue making it challenging for patients to breathe and hinders the transfer of oxygen.1 Patients typically progress to hypoxemia and respiratory failure, with most patients dying from the disease within five years of diagnosis.

ILD can arise due to various acquired or environmental factors, but many genetic etiologies of the disease have been identified.

High-resolution computed tomography (HRCT) of the chest in patients with idiopathic pulmonary fibrosis shows interstitial fibrosis.1 When expert HRCT review is not definitive, patients are referred to lung biopsy for diagnosis. Patients with idiopathic pulmonary fibrosis have lung pathology characterized by interstitial fibrosis, honeycomb changes, fibroblastic foci, and a paucity of inflammation. HRCT of the chest alone is not capable of differentiating patients with some lysosomal disorders from patients with other ILDs.

Genetic causes of ILD are heterogeneous thereby making it difficult for a physician to assign a definite genetic diagnosis based on the clinical presentation. This panel includes genes associated with familial pulmonary fibrosis, short telomere syndrome, and surfactant proteins as well as other syndromic genetic conditions.

Incidence:

  • The incidence of idiopathic pulmonary fibrosis is approximately 20 in 100,000 males and 13 in 100,000 females.1
  • The global prevalence of ASMD is approximately 0.5 in 100,000 births.4
Request a kit Test requisition form Sample collection instructions Ordering resources
the lantern project

Program eligibility

This program is for patients in the US who have a clinical diagnosis or radiographic evidence of interstitial lung abnormality in which the underlying cause is undiagnosed (including idiopathic pulmonary fibrosis, progressive pulmonary fibrosis, familial pulmonary fibrosis).

About the test

  • NGS gene panel will include genes associated with familial and idiopathic ILD, genes associated with surfactant and surfactant proteins, genes associated with telomere disorders, and genes where ILD is part of a larger multisystem disorder. Additionally, glucocerebrosidase enzyme analysis will be run in parallel.
  • If a blood sample (as opposed to saliva) is submitted, enzyme assay for glucocerebrosidase (Gaucher) will be performed in parallel due to complexities with GBA1 sequencing by NGS.
  • If a variant in GLA is found (whether pathogenic, likely pathogenic or variant of uncertain significance), this test will reflex to α-galactosidase A enzyme analysis and lyso-Gb3 analysis.
  • If two variants in SMPD1 are found (whether pathogenic, likely pathogenic or variants of uncertain significance), this test will reflex to acid sphingomyelinase enzyme analysis.
  • If glucocerebrosidase enzyme assay is low, this test will reflex to GBA1 analysis and lyso-Gb1 testing.

Sample requirements

Enzyme assay: Dried blood spots are preferred, but whole blood is also acceptable.

Gene sequencing: Dried blood spots (DBS) are preferred, but whole blood is also acceptable. A saliva sample can be used if only gene sequencing is being ordered.

Bundled testing (Enzyme assay with reflex to sequencing and biomarker): Dried blood spots (DBS) are preferred, but whole blood is also acceptable. A saliva sample cannot be used for enzyme assay or biomarker measurement.

Methodology

Panel methodology:

Sequencing is performed on genomic DNA using a targeted sequence capture method to enrich for the genes of interest. Direct sequencing of the amplified captured regions is performed using 2X150bp reads on next generation sequencing (NGS) systems. A base is considered to have sufficient coverage at 20X and an exon is considered fully covered if all coding bases plus three nucleotides of flanking sequence on either side are covered at 20X or more. A list of these regions, if any, is available upon request. Alignment to the human reference genome (GRCh37) is performed and annotated variants are identified in the targeted region. Variants reviewed have a minimum coverage of 8X and an alternate allele frequency of 20% or higher. Indel and single nucleotide variants (SNVs) may be confirmed by Sanger sequence analysis before reporting at director discretion. This assay cannot detect variants in regions of the exome that are not covered, such as deep intronic, promoter and enhancer regions, areas containing large numbers of tandem repeats, and variants in mitochondrial DNA. Copy number variation (CNV) analysis detects deletions and duplications; in some instances, due to the size of the exons, sequence complexity, or other factors, not all CNVs may be analyzed or may be difficult to detect. When reported, copy number variant size is approximate. Actual breakpoint locations may lie outside of the targeted regions. CNV analysis will not detect tandem repeats, balanced alterations (reciprocal translocations, Robertsonian translocations, inversions, and balanced insertions), methylation abnormalities, triploidy, and genomic imbalances in segmentally duplicated regions. This assay is not designed to detect mosaicism; possible cases of mosaicism may be investigated at the discretion of the laboratory director. Tertiary data analysis is performed using SnpEff v5.0 and Revvity Omics' internal ODIN v.1.01 software. CNV and absence of heterozygosity are assessed using Bionano’s NxClinical v6.1 software.

GBA1 testing methodology:

Long-range PCR is performed to capture the true genomic sequences for the gene of interest from this individual’s genomic DNA to avoid pseudogene contaminants. PCR product is utilized to prepare for the library and further sequenced using Next-generation sequencing (NGS) with 150 base pair paired-end reads. Indels and SNVs are confirmed by Sanger sequence analysis before reporting at the director’s discretion. This analysis cannot detect variants in regions not analyzed such as promoters, deep intronic regions, or long repetitive regions. Copy number variation (CNV) analysis is assessed using MLPA or manual reviewing of NGS reads using IGV viewer. This analysis cannot determine the location or orientation of a duplication. Copy neutral gene conversion or fusion, complex rearrangement, or large deletion including the entire GBA gene with breakpoint(s) outside the targeted region may not be detected by this assay. This assay is not designed to detect mosaicism; possible cases of mosaicism may be investigated at the discretion of the laboratory director.

Enzyme assay:

Enzyme activity is measured on dried blood spots (DBS) via Flow Injection Tandem Mass Spectrometry (FIA/MS/MS).

Turn-Around-Times (TAT)

Enzyme assay: 3 days

Gene sequencing: 3 weeks

Genes tested

ABCA3, ACVRL1, AP3B1, ATP13A3, BMPR1B, BMPR2, CAV1, CCBE1, COPA, CSF2RA, CSF2RB, DICER1, DKC1, EIF2AK4, ENG, FAM111B, FAT4, FGF10, FLCN, FLNA, FLT4, FOXC2, FOXF1, GATA2, GDF2, GLA, HPS1, HPS4, KCNK3, MARS1, MUC5B, NAF1, NKX2-1, NPC1, NPC2, OAS1, PARN, RTEL1, SERPINA1, SFTPA1, SFTPA2, SFTPB, SFTPC, SLC34A2, SLC7A7, SMAD4, SMAD9, SMPD1, STAT3, STING1, STRA6, TBX4, TERC, TERT, TINF2, ZCCHC8

References

  1. Borie R, Le Guen P, Ghanem M, et al. The genetics of interstitial lung diseases. Eur Respir Rev. 2019;28(153):190053. Published 2019 Sep 25. doi:10.1183/16000617.0053-2019.
  2. Raghu G, Torres JM, Bennett RL. Genetic factors for ILD-the path of precision medicine. Lancet Respir Med. 2024 Mar 20:S2213-2600(24)00071-7. doi: 10.1016/S2213-2600(24)00071-7. Epub ahead of print. PMID: 38521082.
  3. Geberhiwot T, Wasserstein M, Wanninayake S, Bolton SC, Dardis A, Lehman A, Lidove O, Dawson C, Giugliani R, Imrie J, Hopkin J, Green J, de Vicente Corbeira D, Madathil S, Mengel E, Ezgü F, Pettazzoni M, Sjouke B, Hollak C, Vanier MT, McGovern M, Schuchman E. Consensus clinical management guidelines for acid sphingomyelinase deficiency (Niemann-Pick disease types A, B and A/B). Orphanet J Rare Dis. 2023 Apr 17;18(1):85. doi: 10.1186/s13023-023-02686-6. PMID: 37069638; PMCID: PMC10108815.
  4. McGovern MM, Wasserstein MP, Giugliani R, Bembi B, Vanier MT, Mengel E, Brodie SE, Mendelson D, Skloot G, Desnick RJ, Kuriyama N, Cox GF. A prospective, cross-sectional survey study of the natural history of Niemann-Pick disease type B. Pediatrics. 2008 Aug;122(2):e341-9. doi: 10.1542/peds.2007-3016. Epub 2008 Jul 14. PMID: 18625664; PMCID: PMC2692309.

How to order

Step 1

Test selection and place order

Step 2

Specimen collection and shipment

Step 3

Get results
Learn More

How to order

1. Test selection and place order

Select the correct test for your patient, and fill out The Lantern Project Requisition Form.

  • Please make sure that all sections are completed, and that the patient has signed the informed consent form.
2. Specimen collection and shipment
  • Obtain a sample for testing from the patient using one of the provided Revvity Omics test packs. If you do not have a kit available in your office, please contact us here and we can have one sent out to your office.
    • Ensure that the patient sample is labeled with the patient’s name and date of birth.
    • Please note that all biochemical assays require a dried blood spot sample or whole blood. Step-by-step instructions for collecting a sample can be found here.
    • Samples may be submitted without a collection kit by following the guidelines for specimen requirements and completing the requisition form.
  • Package the patient sample, informed consent form, and test requisition form back into the test kit, and utilize the included pre-paid shipping label to return the kit to Revvity Omics for processing.
    • As a patient’s clinical presentation is an essential part of fully interpreting genetic test results, we ask that you kindly include any applicable medical records or clinical notes with the sample at the time of test submission.
3. Get results

Once Revvity Omics receives the sample, you will receive phone call to report abnormal findings, with a written report to follow within the established turnaround time for the ordered test. Bundled tests will be reported together in one comprehensive result.

Close

The Lantern Project is not intended to and should not interfere in any way with a health care professional's or patient's independent judgment and freedom of choice in the treatment options for these diseases. Health care professionals and patients should always consider the full range of treatment options and select those most appropriate for the individual patient.

This testing service has not been cleared or approved by the U.S. Food and Drug Administration. Testing services may not be licensed in accordance with the laws in all countries. The availability of specific test offerings is dependent upon laboratory location. The content on this page is provided for informational purposes only, not as medical advice. It is not intended to substitute the consultation, diagnosis, and/or treatment provided by a qualified licensed physician or other medical professionals.

Revvity Logo

©2025 Revvity - All rights reserved

Revvity is a trademark of Revvity, Inc. All other trademarks are the property of their respective owners.